Researchers have learned how a mutation in the gene for superoxide dismutase 1, which causes ALS, leads cells to accumulate damaged materials. The study suggests a potential target for treating this familial form of ALS.



Source link

Leave a Reply

Your email address will not be published. Required fields are marked *

Explore More

Re-purposed FDA-approved drug could help treat high-grade glioma

High-grade glioma, an aggressive form of pediatric and adult brain cancer, is challenging to treat given the tumor location, incidence of recurrence and difficulty for drugs to cross the blood-brain

Squid studies suggest new route to therapy for ALS, targeting synaptic dysfunction

Amyotrophic lateral sclerosis (ALS) is one of the most devastating adult-onset neurodegenerative diseases. Patients, including the late actor/playwright Sam Shepard, become progressively weaker and eventually paralyzed as their motor neurons

Administration of steroid to extremely preterm infants not associated with adverse effects on neurodevelopment

The administration of low-dose hydrocortisone to extremely preterm infants was not associated with any adverse effects on neurodevelopmental outcomes at 2 years of age, according to a study published by